The best answer is C.

Here's Why: All peer-reviewed studies relating to survival of sudden cardiac arrest identify electrical therapy (early defibrillation) as the most successful intervention associated with overall response and survival.

The best answer is C.

Here's Why: Atropine does not affect adrenergic receptors like catecholamine drugs (epinephrine, dopamine, etc.) do. Atropine is a parasympatholytic agent that may speed heart rate due to its effect on the vagas nerve. Reasonable caution should be used when administering atropine as unwanted tachycardia can occur. Not all patients with symptomatic bradycardias will respond to atropine due to infra-nodal lesions or ventricular escape mechanisms.

The best answer is C.

Here's Why: The most reproducible response of young children to clinically significant hypoxemia is worsening bradycardia. Bradycardia in unstable pediatric patients should always be first addressed by providing an open airway and supplemental oxygen with assisted ventilation if required.

The best answer is D.

Here's Why: Changes at the sinus node, 1st degree AVB, 2nd degree AVB type I (AV node level lesions) are most often caused by transient and reversal events. 2nd degree AVB type II is most commonly caused by an infranodal lesion and is most often organic in nature. Infranodal lesions typically require placement of transcutaneous (emergency) or transvenous cardiac pacemakers.

The best answer is B.

Here's Why: According to the most recent recommendations of the International Liaison Committee on Resuscitation (ILCOR), oxygen should only be administered to these patients if there is any sign of respiratory difficulty or hypoxemia. Patients who have stable Sa02’s with no signs of hypoxemia should not receive “prophylactic oxygen therapy.”